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And Robakis, N. K. (1998). Alzheimer's disease: A re-examination of the amyloid hypothesis. Trends Neurosci. 21, 15-19. Newell, K. , Hyman, B. , Growdon,J. , and Hedley-Whyte, T. E. (1999). J. Neuropathol. Exp. Nenrol. 58, 1147-1155. , and Wang, X. (2000). Apoptosis in neural development and disease. Annu. Rev. Neurosci. 23, 73-87. , and Nakano, M. (1990). Estrogens as antioxidants. Methods Enzymol. 186, 330-333. Ohm, T. , and Bohl, J. (1997). Unbiased estimation of neuronal numbers in the human nucleus coeruleus during aging.

Staging of Alzheimer-type pathology: An interrater-intrarater study. Dement. Geriatr. Cogn. Disord. 8, 248-251. , Yanker, B. , and Yuan,J. (2000). Caspase12 mediates endoplasmic reticulum-specific apoptosis and cytotoxicity by amyloid-fl. Nature 403, 98-103. Nathan, B. , Sanan, D. , Weisgraber, K. , Mahley, R. , and Pitas, R. E. (1994). Differential effects of apolipoproteins E3 and E4 on neuronal growth in vitro. Science264, 850-852. National Institute on Aging and Reagan Institute Working Group on Diagnostic Criteria for the Neuropathological Diagnosis of Alzheimer's Disease.

2, 93-97. Curran, M.. , and Neill, D. (1997). HLA-DR antigens associated with major genetic risk for late-onset Alzheimer's disease. Neuroreport 8, 1467-1469. , and Pradier, L. (2000). Presenilins and Alzheimer's disease: Biological functions and pathogenic mechanisms. Prog. Neurobiol. 60, 363-384. de la Torre, J. C. (1997). Hemodynamic consequences of deformed microvessels in the brain in Alzheimer's disease. Ann. N. E Acad. Sci. 826, 75-91. de la Torre,J. C. (2000). Critically attained threshold of cerebral hypoperfusion: The CATCH hypothesis ofAlzheimer's pathogenesis.

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