Download Pancreatology: From Bench to Bedside by A. K. Saluja, V. Dudeja (auth.), Juan Iovanna, Uktam PDF

By A. K. Saluja, V. Dudeja (auth.), Juan Iovanna, Uktam Ismailov (eds.)

'Pancreatology: from Bench to Bedside' focusses on contemporary advances in pancreatic easy examine, genetics of the pancreatic illnesses and its surgical administration.

Basic study discusses the implication of pancreatic pressure protein in acute pancreatitis and pancreatic melanoma and their attainable position as healing goals. additionally, very unique effects convey the unforeseen function of lipids as mediators in the course of acute pancreatitis. Gene screening thoughts let the detection of the genes responsbile for gemcitabine resistance of pancreatic melanoma cells . They result in the choice of a number of aim genes, with a view to suppress the resistance of cells to gemcitabine therapy. The mechanism through which tetrahydrocannabinol is anti-tumoral in pancreatic melanoma cells is gifted and using THC as a promising new healing agent is mentioned.

Genetic facts are proven touching on 1000's of households with hereditary power pancreatitis and their attainable position within the pathogenesis of the ailment. one other very unique learn addresses the prevention and remedy of pancreatic illnesses with diat.

In medical learn, convincing info concerning the use of endoscopic sphicterotomy within the administration of acute bilary pancreatitis is gifted, in response to the event of a middle hugely really good in pancreatic ailments.

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Pancreatology: From Bench to Bedside

'Pancreatology: from Bench to Bedside' focusses on fresh advances in pancreatic easy learn, genetics of the pancreatic illnesses and its surgical administration. simple examine discusses the implication of pancreatic pressure protein in acute pancreatitis and pancreatic melanoma and their attainable function as healing objectives.

Extra info for Pancreatology: From Bench to Bedside

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2007;3: 145–148. 68. Webber JL, Young AR, Tooze SA. Atg9 trafficking in Mammalian cells. Autophagy. 2007;3: 54–56. 69. Young AR, Chan EY, Hu XW, Kochl R, Crawshaw SG, High S, Hailey DW, LippincottSchwartz J, Tooze SA. Starvation and ULK1-dependent cycling of mammalian Atg9 between the TGN and endosomes. J Cell Sci. 2006;119:3888–3900. 70. Aho HJ, Nevalainen TJ. Experimental pancreatitis in the rat Ultrastructure of sodium taurocholateinduced pancreatic lesions. Scand J Gastroenterol. 1980;15:417–424.

2001, 50:1771–1777. 19. Dersch K, Ichijo H, Bhakdi S, Husmann M. Fatty acids liberated from low-density lipoprotein trigger endothelial apoptosis via mitogen-activated protein kinases. Cell Death Differ. 2005, 12:1107–1114. 32 D. Closa et al. 20. Rizzo G, Fiorucci S. PPARs and other nuclear receptors in inflammation. Curr Opin Pharmacol. 2006, 6:421–427. 21. Kliewer SA, Sundseth SS, Jones SA, Brown PJ, Wisely GB, Koble CS, Devchand P, Wahli W, Willson TM, Lenhard JM, Lehmann JM. Fatty acids and eicosanoids regulate gene expression through direct interactions with peroxisome proliferator-activated receptors alpha and gamma.

Nevertheless, there are lipid mediators that play a role in the regulation of the cell response to cytokines and other proinflammatory mediators. It has long been known that macrophage functions and gene expression can be modulated by specific nuclear receptor pathways, and some of them are under the control of lipid mediators. In particular, PPARs are nuclear receptors activated by fatty acid derivatives. PPARg is one of the members of the PPAR family that has gained interest as it is been involved in the modulation of the inflammatory response [25].

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